As a member of a major trauma unit, it's amazing how many cyclists we see each week- often really competent amateur racers, rarely the dainty commuters. So I suspect, if Pro cycling is counted as an occupation (which is certainly should be- it's how they earn their money), then it would definitely be in the top 10 most risky occupations. Looking at the above posted web page, most of that top 10 list are in the realms of 10% injury rate. Cycling is way above that, with well over 16 of the 160-200-odd starters of a Grand Tour having suffered a crash related injury that year.
Secondly, the cause for poor Kim Kirchen's "collapse" is a mystery to us all right now. In response to my esteemed colleague's informative post re: potential causes, I'm not sure about the hypertrophic cardiomyopathy differential. Ventricular hypertrophy obviously does occur in athletes. However, the etiology is very different to the hypertension patient. In elite rowers for instance, the significant hypertrophy exists with preservation of the ventricular lumen size. the theory being that high intrathoracic pressure generated during the initial "curled up" catch phase of the stroke plus strong muscle contraction alters the way the muscle hypertrophies. This position is very similar to cycling in the drops or TT and I would assume a similar mechanism. If I dig up the reference, I'll post it. What a great topic for a work-related "conference" in some ski resort or Mallorca in summer!
I agree, PE is less likely as is AMI. My money is on the same arrythmia problems he had previously. As was mentioned, this fits with a sudden collapse.
What ever the cause, the fact is that this poor fellow is in a lot of strife medically and the last thing anyone needs (ie. his family, the people who count here) is the instant conclusion that every collapse equals doping.
Secondly, the cause for poor Kim Kirchen's "collapse" is a mystery to us all right now. In response to my esteemed colleague's informative post re: potential causes, I'm not sure about the hypertrophic cardiomyopathy differential. Ventricular hypertrophy obviously does occur in athletes. However, the etiology is very different to the hypertension patient. In elite rowers for instance, the significant hypertrophy exists with preservation of the ventricular lumen size. the theory being that high intrathoracic pressure generated during the initial "curled up" catch phase of the stroke plus strong muscle contraction alters the way the muscle hypertrophies. This position is very similar to cycling in the drops or TT and I would assume a similar mechanism. If I dig up the reference, I'll post it. What a great topic for a work-related "conference" in some ski resort or Mallorca in summer!
I agree, PE is less likely as is AMI. My money is on the same arrythmia problems he had previously. As was mentioned, this fits with a sudden collapse.
What ever the cause, the fact is that this poor fellow is in a lot of strife medically and the last thing anyone needs (ie. his family, the people who count here) is the instant conclusion that every collapse equals doping.