For the Giro 2011:
So his HT declined during the GT, as it should, and his retics went up, which might be in response to the natural decline of the HT. The only thing suspicious during this GT is the consistently high retic levels, as I noted before. The final measurement, showing a fairly high HT, came after the GT. Unless he was preparing for another race (anyone know?), there would be no reason to transfuse at this point. So maybe that HT was natural, with the rise resulting from the recovery after the GT.
TDF 2011:
HT stable during the Tour, it would be expected to fall, but at least it didn’t rise. And again, a rise after the GT, which might have been from natural recovery. Retics did rise during the GT, though, which could certainly indicate EPO use, preventing the natural fall in HT.