hiero2 said:
Have you ever trained to the absolute peak of your cardio ability? It may not be true for everyone - but a lot of cyclists who do find themselves MORE prone to little infections - not less. And little infections can hurt performance. I found this to be true back when I was racing. I find nothing dramatic to doubt in this announcement and move by Cav.
On the other hand, if he suddenly announces he has had asthma for all these years - and requires an inhaler? I would find some grounds for a little doubting.
Otherwise, methinks you are overdramatizing the bronchial thing.
I do. I don't have a susceptibility to any infections of any type related to chest/colds etc..in fact, I rarely get them.
With that being said. There have been studies showing a suppression of immunity after extreme exertions/levels of high exercise.
Varies by person though.
Blurb from an article I lifted:
"Post-exercise immune function depression is most pronounced when the exercise is continuous, prolonged (>1.5 hours), of moderate to high intensity (55-75% of aerobic capacity), and performed without food intake (Gleeson, 2006). Both aspects of innate immunity (e.g., neutrophil chemotaxis, phagocytosis, degranulation and oxidative burst activity and natural killer cell cytoxic activity) and acquired immunity (e.g., toll-like receptor expression and antigen presentation by monocytes/ macrophages; T lymphocyte cytokine production and proliferation, immunoglobulin production by B lymphocytes) are depressed by prolonged exercise. The salivary secretory immunoglobulin A (SIgA) response to acute exercise is variable, though very prolonged bouts of exercise are commonly reported to result in decreased SIgA secretion (Bishop & Gleeson, 2009). The causes of immune depression after prolonged exercise are thought to be related to increases in circulating stress hormones (e.g., adrenaline and cortisol), alterations in the pro/antiinflammatory cytokine balance and increased free radicals.
Markers of immune function in athletes in the true resting state (i.e., at least 24 hours after the last exercise bout) are generally not very different from their sedentary counterparts, except when athletes are engaged in periods of intensified training. In this situation immune function might not fully recover from successive training sessions and some functions can become chronically depressed (Gleeson, 2005). Both T and B lymphocyte functions appear to be sensitive to increases in training load in well-trained athletes undertaking a period of intensified training, with decreases in circulating numbers of Type 1 T cells, inhibition of Type 1 T cell cytokine production, reduced T cell proliferative responses and falls in stimulated B cell immunoglobulin synthesis and SIgA reported. However, to date, the only immune variable that has been associated with increased infection incidence is SIgA. Low concentrations of SIgA in athletes or substantial transient falls in SIgA are associated with increased risk of URTI. In contrast, increases in SIgA can occur after a period of regular moderate exercise training in previously sedentary individuals and could, at least in part, contribute to the apparent reduced susceptibility to URTI associated with regular moderate exercise compared with a sedentary lifestyle."
BUT......before you jump to conclusion about that study, in general, there is very little evidence to show exercise is the exact cause/reason for infections in athletes, or makes them more susceptible.
Exercise and Immune Function
Recent Developments
David C. Nieman1 and Bente K. Pedersen2
"Comparison of immune function in athletes and nonathletes reveals that the
adaptive immune system is largely unaffected by athletic endeavour. The innate
immune system appears to respond differentially to the chronic stress of intensive
exercise, with natural killer cell activity tending to be enhanced while neutrophil
function is suppressed. However, even when significant changes in the level and
functional activity of immune parameters have been observed in athletes,
investigators
have had little success in linking these to a higher incidence of infection
and illness."